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Serotonin-containing neurons are present in chick embryos from the time of fertilization until before hatching, and this persistent sensory innervation is thought to be responsible for the posthatching elevation in nociception. However, the role of other neurotransmitters and neuromodulators in mediating this late sensory maturation is not known. Histamine is a neurotransmitter in the developing nervous system, and its presence in embryonic sensory neurons is well established. Here we demonstrate ectodermally derived histamine in chick embryos and demonstrate that this peptidergic nerve is located within nerve fibers innervating the chick embryonic vascular network. We show that this nerve is colocalized with a marker for sensory neurons (Nos1) and mediates vasodilation in a manner comparable to norepinephrine and serotonin. Furthermore, we demonstrate that histamine mediates nociception in the chick embryo and that this is significantly less than that seen in older animals. Our data suggest that histamine may play an important role in mediating the later development of nociceptive sensation in the chick embryo.Posterior spinal instrumentation with use of the K-wire and Chamberlain disk for treating adult spinal deformities.
To introduce a new technique for treating adult spinal deformity. We report our first 9 patients treated with this system. The new technique uses a modified K-wire and Chamberlain disk system for treatment of adult spinal deformity. Description of the new system for treating adult spinal deformity. An older osteoporotic patient, patients with degenerative kyphosis, or patients with dynamic scoliosis are candidates for
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